Show simple item record

dc.contributor.advisorO'Neill, Luke
dc.contributor.authorKenny, Elaine
dc.date.accessioned2019-11-07T15:58:04Z
dc.date.available2019-11-07T15:58:04Z
dc.date.issued2010
dc.identifier.citationElaine Kenny, 'Mal is not esential for TLR2 signalling and inhibits signalling by TLR3', [thesis], Trinity College (Dublin, Ireland). School of Biochemistry and Immunology, 2010, pp 285
dc.identifier.otherTHESIS 9434
dc.description.abstractThis thesis set out to systematically analyse the role of Mai in signalling by TLR2, TLR3 and TLR4 in murine macrophages and dendritic cells. It has revealed that Mai is not essential to TLR2 signal transduction, is required for TLR4 signalling and is inhibitory for signalling by TLR3. The absolute requirement for MyD88 in TLR2 and TLR4 signalling was also confirmed.
dc.format1 volume
dc.language.isoen
dc.publisherTrinity College (Dublin, Ireland). School of Biochemistry and Immunology
dc.relation.isversionofhttp://stella.catalogue.tcd.ie/iii/encore/record/C__Rb14881933
dc.subjectBiochemstry, Ph.D.
dc.subjectPh.D. Trinity College Dublin.
dc.titleMal is not esential for TLR2 signalling and inhibits signalling by TLR3
dc.typethesis
dc.type.supercollectionthesis_dissertations
dc.type.supercollectionrefereed_publications
dc.type.qualificationlevelDoctoral
dc.type.qualificationnameDoctor of Philosophy (Ph.D.)
dc.rights.ecaccessrightsopenAccess
dc.format.extentpaginationpp 285
dc.description.noteTARA (Trinity’s Access to Research Archive) has a robust takedown policy. Please contact us if you have any concerns: rssadmin@tcd.ie
dc.identifier.urihttp://hdl.handle.net/2262/90272


Files in this item

Thumbnail
Thumbnail

This item appears in the following Collection(s)

Show simple item record