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dc.contributor.advisorLawler, Mark
dc.contributor.authorSpellacy, Neil
dc.date.accessioned2017-06-28T11:06:36Z
dc.date.available2017-06-28T11:06:36Z
dc.date.issued2005
dc.identifier.citationNeil Spellacy, 'An investigation into the potential use of Beta-Galactoside binding protein as a novel anti-leukaemic agent', [thesis], Trinity College (Dublin, Ireland). School of Medicine. Discipline of Haematology, 2005, pp 249
dc.identifier.otherTHESIS 7607
dc.description.abstractChronic myelogenous leukaemia (CML) is characterised at the molecular level by a (9;22) translocation which places the abl proto-oncogene under the control of the breakpoint cluster region (bcr) gene promoter generating a fusion protein (p210) with enhanced tyrosine kinase activity. CML cells are inherently resistant to apoptosis induced by conventional chemotherapeutic agents.
dc.format1 volume
dc.language.isoen
dc.publisherTrinity College (Dublin, Ireland). School of Medicine. Discipline of Haematology
dc.relation.isversionofhttp://stella.catalogue.tcd.ie/iii/encore/record/C__Rb12446076
dc.subjectHaematology and Oncology, Ph.D.
dc.subjectPh.D. Trinity College Dublin
dc.titleAn investigation into the potential use of Beta-Galactoside binding protein as a novel anti-leukaemic agent
dc.typethesis
dc.type.supercollectionthesis_dissertations
dc.type.supercollectionrefereed_publications
dc.type.qualificationlevelDoctoral
dc.type.qualificationnameDoctor of Philosophy (Ph.D.)
dc.rights.ecaccessrightsopenAccess
dc.format.extentpaginationpp 249
dc.description.noteTARA (Trinity’s Access to Research Archive) has a robust takedown policy. Please contact us if you have any concerns: rssadmin@tcd.ie
dc.identifier.urihttp://hdl.handle.net/2262/80517


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