Bridging the gap – a new role for STAT3 in TLR4-mediated metabolic reprogramming

dc.contributor.author	O'Neill, Luke
dc.date.accessioned	2021-02-25T15:40:20Z
dc.date.available	2021-02-25T15:40:20Z
dc.date.issued	2021
dc.date.submitted	2021	en
dc.identifier.citation	Weiss, H.J. and O'Neill, L.A.J., Bridging the gap – a new role for STAT3 in TLR4-mediated metabolic reprogramming, Immunology and Cell Biology, 99, 2, 2021, 122-125	en
dc.identifier.other	Y
dc.description.abstract	In this article, we discuss a new study from the laboratory of Ashley Mansell that successfully links innate immune receptor signaling to metabolic changes in macrophages, establishing for the ﬁrst time an uninterrupted pathway from ligand binding to mitochondrial reprogramming and subsequential effector function modulation in this cell type.	en
dc.format.extent	122-125	en
dc.language.iso	en	en
dc.relation.ispartofseries	Immunology and Cell Biology;
dc.relation.ispartofseries	99;
dc.relation.ispartofseries	2;
dc.rights	Y	en
dc.subject	ligand binding	en
dc.subject	macrophages	en
dc.subject.lcsh	ligand binding	en
dc.subject.lcsh	macrophages	en
dc.title	Bridging the gap – a new role for STAT3 in TLR4-mediated metabolic reprogramming	en
dc.type	Journal Article	en
dc.type.supercollection	scholarly_publications	en
dc.type.supercollection	refereed_publications	en
dc.identifier.peoplefinderurl	http://people.tcd.ie/laoneill
dc.identifier.rssinternalid	224565
dc.identifier.doi	http://dx.doi.org/10.1111/imcb.12414
dc.rights.ecaccessrights	openAccess
dc.identifier.uri	http://hdl.handle.net/2262/95390

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